Schematic showing how antisense oligonucleotides restore FKTN protein and α-DG functions in cell-based experiments (IMAGE)

Fujita Health University

Schematic showing how antisense oligonucleotides restore FKTN protein and α-DG functions in cell-based experiments

Caption

RNAi-based treatment restores normal FKTN protein production and alpha-dystroglycan glycosylation in cells derived from patients with Fukuyama Muscular Dystrophy carrying the deep intronic variant

Credit

Mariko Taniguchi-Ikeda from Fujita Health University

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