RIP3-mediated OA pathogenesis via perturbing the bone-cartilage unit (IMAGE)
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RIP3 plays a pivotal role in the pathogenesis of OA by disrupting cartilage extracellular matrix (ECM) metabolism homeostasis, inhibiting bone formation, and enhancing bone resorption, while clofibrate appears to be a promising drug against OA by targeting RIP3.
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